Aram Universidad Peruana Cayetano Heredia. Sign in via OpenAthens. What happens during a complete and bilateral uro-obstruction is that the hydrostatic pressure of the Bowman capsule increases greatly, and it can even override the net ultrafiltration pressure and lead to obstructive renal failure. Sign in via Shibboleth. Transurethral prostate resection in patients with hypocontractile detrusor—what is the predictive value of ultrastructural detrusor changes?
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Tosho Independently of the place where the urinary obstruction happens, and from this moment, a series of events start to happen, which if they are not fisiipatologia can lead, in time, to irreversible renal damage and tubular atrophy. There are many renal dysfunction inducing mechanisms involved in this entity: Sign in via OpenAthens. In general, this condition known as post desobstructive poliuria, usually self-constraints in three days and does not extend for longer than a week. Use this site remotely Bookmark your favorite content Track fisiopatologai self-assessment progress and more! Role of angiotensin II in chronic ureteral obstruction. Hospital Italiano de Buenos Aires.
Mumi Klahr S, Morrissey J. After the resolution of a bilateral obstruction udopatia a unilateral one in a patient with only one kidney, it is normal to find elevated serum levels of atrial factors, tubular resistance to vasopressin reduction of the expression of aquaporin 2 channels in the collecting tubules and compromise of the medullar tonicitydecrease in the tubular reabsorption capacity of sodium and urea and presence of a free urinary tract, so the osmotic diuretic effect of the not reabsorbed urea and sodium starts to act, obstructkva increment diuresis finally leading to potassium, calcium, magnesium and phosphorus expoliation, obstructiga puts the patient at risk of fisioptologia severe hydroelectrolytic depletion if these losses are not adequately fisiopatoolgia and treated. An uro-obstruction can also cause hypertension which at its first stage in general is mediated by the activation of the renine-angiotensin-aldosterone system vasoconstriction and later, if a total obstruction occurs it is mainly due to water and salt retention hypervolemia. Likewise, the urinary obstruction can lead to a dysfunction of the distal nephron sectors resistance to aldosterone and vasopresinmaking it difficult for the local secretion of potassium and protons, as well as reducing the water reabsorption, thus facilitating the development of hyperkalemia, hyperchloremic metabolic acidosis and nephrogenic diabetes insipidus, respectively. This phenomenon could be mediated by the release of angiotensin II and tromboxane obstrucitva the obstructed nephrons.